Tuesday, November 1, 2016

General Health: Eating Disorders - Bulimia nervosa Treatments In Conventional Medicine Perspective

Kyle J. Norton(Scholar, Master of Nutrients), all right reserved.
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.


                               Eating disorders

Eating Disorders are defined as a group of abnormal eating habits associated to a person preoccupation weight, involving either insufficient or excessive food intake.

                                Bulimia nervosa

Bulimia nervosa is defined as a medical condition of consuming a large amount of food in a short amount of time or one setting (binge eating), followed by self induced vomiting, taking a laxative or diuretic and/or excessive exercise, etc. to compensate for the binge. Bulimia nervosa also effects almost 90% of female. Unlike anorexia nervosa, people suffering from bulimia nervosa are usually normal or slightly over weight.

                    The Treatments

In Conventional Medicine Perspective
A.1. Non medication Therapies
1. Group therapy
In the study to evaluate the Guided self-help versus cognitive-behavioral group therapy in the treatment of bulimia nervosa, showed that A mixed-effects linear regression analysis indicated that subjects in both treatment conditions showed a significant decrease over time in binge eating and vomiting frequencies, in the scores of the EDI subscales, and in the BDI. Both treatment modalities led to a sustained improvement at follow-up. A separate analysis of the completer sample showed significantly higher remission rates in the self-help condition (74%) compared with the CBT condition (44%) at follow-up(45).
.
2. Cognitive behavioral guided self-help
In the study of 123 individuals (mean age = 37.2; 91.9% female, 96.7% non-Hispanic White) were randomized, including 10.6% with bulimia nervosa (BN), 48% with binge eating disorder (BED), and 41.4% with recurrent binge eating in the absence of BN or BED. Baseline, posttreatment, and 6- and 12-month follow-up data, showed that Cognitive behavioral guided self-help is a viable first-linetreatment option for the majority of patients with recurrent binge eating who do not meet diagnostic criteria for BN or anorexia nervosa(46).

3. Psychoeducational therapy
In the assessment of 241 seeking-treatment females with bulimia nervosacompleted an exhaustive assessment and were referred to a six-sessionpsychoeducational group, Regression analyses of treatment response were performed. Childhood obesity, lower frequency of eating symptomatology, lower body mass index, older age, and lower family's and patient's concern about the disorder were predictors of poor abstinence. Suicidal ideation, alcohol abuse, higher maximum BMI, higher novelty seeking and lower baseline purging frequency predicted dropouts. Predictors of early symptom changes and dropouts were similar to those identified in longer CBT interventions(47).

4. Psychodynamic therapy
In the examined 14 bulimic clients' experiences of individual psychodynamicpsychotherapy through semistructured interviews, which were analyzed using qualitative methods. The results showed that the psychodynamic approach was a challenge to most of the clients. Yet, most clients profited from therapy both symptomatically and with regard to interpersonal relations and affect regulation. There were, however, marked differences in the clients' experiences. One subgroup rather quickly felt that the therapy met their needs, another initially felt challenged by the approach and the therapeutic attitude but ultimately succeeded in using this particular kind of therapy. A third group remained predominantly critical of their therapies. The clinical implications and possible explanations of the results are discussed(48).


5. Relational theory
In the article to explain how the psychology of women can inform group treatment by translating relational theory (RT) into practice within a short-term outpatientbulimia group. First, the article provides a brief overview of a relationalunderstanding of women's psychological development, the etiology and maintenance of bulimia nervosa, and group psychotherapy. Then, clinical vignettes illustrate the application of RT in practice through discussion of four main healing factors at work in the different stages of the group. Through promoting validation, self-empathy, mutuality, and empowerment, the leader helps group members identify and change relational patterns that have kept them connected with food and disconnected from themselves and others. The goal of treatment is to help members move toward mutually empathic and empowering relationships inside and outside the group(49).

6. Cognitive-Behavioral therapy(CBT)
In the study to examine the potential efficacy of CBT for eating disorder individuals with bulimic symptoms who do not meet full criteria for bulimia nervosa. Twelve participants with subthreshold bulimia nervosa were treated in a case series with 20 sessions of CBT. Ten of the 12 participants (83.3%) completed treatment. Intent-to-treat abstinent percentages were 75.0% for objectively large episodes of binge eating (OBEs), 33.3% for subjectively large episodes of binge eating (SBEs), and 50% for purging at end of treatment. At one year follow-up, 66.7% were abstinent for OBEs, 41.7% for SBEs, and 50.0% for purging(50).

7. Etc.

A.2. Medical treatments
Fluoxetine (Prozac), a type of selective serotonin reuptake inhibitor (SSRI, the only antidepressant approved by the Food and Drug Administration may help to ease the symptoms of bulimia.

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Sources
(45) http://www.ncbi.nlm.nih.gov/pubmed/15101068
(46) http://www.ncbi.nlm.nih.gov/pubmed/20515207
(47) http://www.ncbi.nlm.nih.gov/pubmed/19501787
(48) http://www.ncbi.nlm.nih.gov/pubmed/21198236
(49) http://www.ncbi.nlm.nih.gov/pubmed/9766090
(50) http://www.ncbi.nlm.nih.gov/pubmed/22290037

Monday, October 31, 2016

General Health: Eating Disorders - Bulimia nervosa - The Antioxidants

Kyle J. Norton(Scholar, Master of Nutrients), all right reserved.
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.


                               Eating disorders

Eating Disorders are defined as a group of abnormal eating habits associated to a person preoccupation weight, involving either insufficient or excessive food intake.

                                Bulimia nervosa

Bulimia nervosa is defined as a medical condition of consuming a large amount of food in a short amount of time or one setting (binge eating), followed by self induced vomiting, taking a laxative or diuretic and/or excessive exercise, etc. to compensate for the binge. Bulimia nervosa also effects almost 90% of female. Unlike anorexia nervosa, people suffering from bulimia nervosa are usually normal or slightly over weight.

                    The  Antioxidants 

Please read Antioxidant to prevent anorexia nervosa
http://kylejnorton.blogspot.ca/2013/12/anorexia-nervosa-antioxidants.html

Friday, October 28, 2016

General Health: Eating Disorders - Bulimia nervosa - The Diet and nutritional supplements

Kyle J. Norton(Scholar, Master of Nutrients), all right reserved.
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.


                               Eating disorders

Eating Disorders are defined as a group of abnormal eating habits associated to a person preoccupation weight, involving either insufficient or excessive food intake.

                                Bulimia nervosa

Bulimia nervosa is defined as a medical condition of consuming a large amount of food in a short amount of time or one setting (binge eating), followed by self induced vomiting, taking a laxative or diuretic and/or excessive exercise, etc. to compensate for the binge. Bulimia nervosa also effects almost 90% of female. Unlike anorexia nervosa, people suffering from bulimia nervosa are usually normal or slightly over weight.

                    The Diet and nutritional supplements

The aim of the diet and nutritional supplements is to provide enough nutrients as for people with anorexia nervosa are more likely to have vitamin and mineral deficiencies which can lead to certain symptoms of the diseases.
1. Caffeine
Caffeine intake increased over time between ages 9 and 19 years across all groups and this trend was not moderated by diagnostic status. For anorexia nervosa, relative to the non-eating disorder group, the proportional intake ofcaffeine from soda increased significantly before onset to onset to after onset and ingestion of chocolate-containing foods decreased sharply over time(42).

2. Alcohol
While the rate of anorexia was not elevated in alcoholics after controlling for other disorders, bulimia did occur at a greater than expected rate. However, both eating disorders were relatively rare, and much of the association with alcoholism occurred in the context of additional preexisting or secondary psychiatric disorders(43).

3. Tobacco
Although malnutrition may be expected to reduce DNA methylation through its effects on one-carbon metabolism, our negative results are in line with several in vitro and clinical studies that did not show a direct relation between gene-specific DNA methylation and folate levels. In contrast, smoking has been repeatedly reported to alter DNA methylation of specific genes and should be controlled for in future epigenetic studies(44)
.
4. Drink 6 - 8 glasses of filtered water daily as water can decrease the risk of dehydration.
Caffeine, water, and aspartame consumption can be variable in patients with AN and the consumption of these substances seems to be only modestly related to purging behavior(45).

5. Promote healthy diet for maximum nutrients absorption.
6. The important of nutritional supplements
Some researchers suggested that conservation mechanisms resulting from starvation and/or self-prescribed nutrient supplements can result in laboratory values that appear within normal limits. These artificially inflated values drop to dangerous levels in some patients once rehydration and refeeding begin. Electrolyte status must be closely monitored during this time to prevent complications. Other micronutrient deficiencies can be corrected with adequatedietary intake, but patients with eating disorders are unlikely to consume such an adequate diet immediately upon entering treatment, so they may benefit from supplementation. Depleted nutrient stores require longer supplementation than acute inadequacies in nutrient intake. This review compiles the findings reported to date regarding micronutrient deficiencies and supplementation for patients withanorexia and bulimia. Because of the widely varying eating practices from patient to patient and the current lack of data controlling for nutrient self-supplementation, nutrition assessment performed by a nutrition professional via food intake history may be more practical than laboratory tests and more accurate than current food intake for determining potential micronutrient deficiencies(46).
a.. In the study of 20 female patients with anorexia nervosa (AN) and in 10 lean and 10 normal weight, healthy, female control subjects. Patients with AN hadhigher activities of L-gamma-glutamyl transferase (gamma-GT) and glutamate pyruvate transaminase (SGPT) and a higher concentration of prealbumin in serum and lower leucocyte and lymphocyte counts in blood. For the other routine clinical chemical parameters no significant differences between the groups were observed. AN patients had higher serum vitamin B12 and retinol levels. No significant differences were found for the status parameters of thiamin, vitamin B6, vitamin C, folate, vitamin E and vitamin D. Contradictory results were obtained for the riboflavin status: AN patients had a lower level of flavin adenine dinucleotide (FAD) in blood and a lower stimulation ratio of the glutathione reductase activity in erythrocytes (alpha-EGR). Patients with AN had higher serum ferritin concentration and lower total iron binding capacity (TIBC). However, haemoglobin (Hb), haematocrit (Ht) and iron saturation were not significantly different. No significant difference was found in the concentration of zinc in plasma. In spite of the poor intake of nutrients and energy, the results obtained did not indicate an inadequate status of vitamins, iron and zinc in patients with AN(47).

b. Other study of trace metals, vitamins, and other biochemical parameters in 30 female patients hospitalized for anorexia nervosa, showed that Anorexia nervosapatients showed hypogeusia, with the bitter and sour taste most severely affected, however plasma zinc levels did not correlate with taste recognition scores. Patients showed hypercarotenemia (214 +/- 129 microgram/100 ml; P < 0.01) with normal plasma vitamin A and retinol-binding protein levels. Total iron binding capacity was depressed (261 +/- 62 microgram/100 ml; P < 0.001) in contrast to plasma iron, ceruloplasmin and folic acid, which were normal. In nine patients, who were retested before discharge, taste function improved; plasma zinc, copper, and total iron binding capacity levels increased whereas plasma carotene and cholesterol decreased to normal levels. It is concluded that the observed zinc, copper, and iron binding protein deficiencies, and hypogeusia, reflect the self-imposed nutritional restriction of anorexia nervosa patients. Zinc and other micronutrients released from catabolized tissue along with vitamin intake may mitigate against more severe deficiency states in anorexia nervosa(48).
A daily multivitamin is an essential, as it contain numbers of vitamins and trace minerals such as vitamins A, C, E, the B-vitamins, magnesium, calcium, zinc, phosphorus, copper, and selenium which are essential for the body needed. Other supplement include Omega-3 fatty acids, Coenzyme Q10, 5-hydroxytryptophan (5-HTP), Creatine, Probiotic supplement, etc.

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Sources
(42) http://www.ncbi.nlm.nih.gov/pubmed/22133028
(43) http://www.ncbi.nlm.nih.gov/pubmed/20512042
(44) http://www.ncbi.nlm.nih.gov/pubmed/20441789
(45) http://www.ncbi.nlm.nih.gov/pubmed/15101068
(46) http://www.ncbi.nlm.nih.gov/pubmed/20515207
(47) http://www.ncbi.nlm.nih.gov/pubmed/19501787
(48) http://www.ncbi.nlm.nih.gov/pubmed/21198236

General Health: Eating Disorders - Bulimia nervosa - The Do's and Do not's list

Kyle J. Norton(Scholar, Master of Nutrients), all right reserved.
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.


                               Eating disorders

Eating Disorders are defined as a group of abnormal eating habits associated to a person preoccupation weight, involving either insufficient or excessive food intake.

                                Bulimia nervosa

Bulimia nervosa is defined as a medical condition of consuming a large amount of food in a short amount of time or one setting (binge eating), followed by self induced vomiting, taking a laxative or diuretic and/or excessive exercise, etc. to compensate for the binge. Bulimia nervosa also effects almost 90% of female. Unlike anorexia nervosa, people suffering from bulimia nervosa are usually normal or slightly over weight.

                     The Do's and Do not's list

A.1. Primary prevention
Dt. Michael Sidiropoulos in the article of Anorexia Nervosa: The physiological consequences of starvation and the need for primary prevention efforts indicated that there are numerous actions that the physician, along with the family, allied health care workers and/or through a broader public health initiative can accomplish in this particular case that will have longstanding implications on the patient's future development and growth and will increase the likelihood of healthy outcomes through primary
prevention(37).
1. Minimizing social pressures
In the study to evaluation the Sociological factors in the development of eating disorders, Dr Nagel KL, andand Dr. Jones KH. at the University of Georgia indicated that professionals in the educational and physical and mental health care fields need to be aware of the influence of social pressures on teenagers' perceptions of body image and appearance. This article reviews the sociocultural, socioeconomic, and sex-related factors which contribute to the development of eating disorders. It is recommended that professionals help adolescents resist societal pressure to conform to unrealistic standards of appearance, and provide guidance on nutrition, realistic body ideals, and achievement of self-esteem, self-efficacy, interpersonal relations and coping skills(38).

2. Minimizing family issues
Dr. Yager J. in the study of the family issues in the pathogenesis of anorexia nervosa, suggested that factors residing in family systems have been implicated in the pathogenesis of anorexia nervosa. In this paper I critically review literature that bears on this issue: the transmission of anorexia nervosa in families; familystress patterns, personality and psychopathological characteristics of parents, parent-child interactions, and whole family systems. Much additional research is needed to accurately determine the precise nature of such factors and the extent to which they actually contribute to the appearance of this syndrome(39).

3. Reducing individual factors
In the study to examine which unique factors (genetic and environmental) increase the risk for developing anorexia nervosa by using a case-control design of discordant sister pairs, Dr. Karwautz A, and the research team at University of London, suggested that he sisters with anorexia nervosa differed from their healthy sisters in terms of personal vulnerability traits and exposure to high parental expectations and sexual abuse. Factors within the dieting risk domain did not differ. However, there was evidence of poor feeding in childhood. No difference in the distribution of genotypes or alleles of the DRD4, COMT, the 5HT2A and 5HT2C receptor genes was detected. These results are preliminary because our calculations indicate that there is insufficient power to detect the expected effect on risk with this sample size(40).

A.2. Secondary prevention
Secondary prevention focuses early detection and intervention as early detection is often difficult as individuals with eating problems often attempt to conceal their behavior. People such as Parents, peers and siblings, teacher and family doctor are in good position to detect changing attitudes around food,weight, and shape for detecting eating disorder early for effective treatment(41).

Thursday, October 27, 2016

General Health: Eating Disorders - Bulimia nervosa - The Diagnosis

Kyle J. Norton(Scholar, Master of Nutrients), all right reserved.
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.


                               Eating disorders

Eating Disorders are defined as a group of abnormal eating habits associated to a person preoccupation weight, involving either insufficient or excessive food intake.

                                Bulimia nervosa

Bulimia nervosa is defined as a medical condition of consuming a large amount of food in a short amount of time or one setting (binge eating), followed by self induced vomiting, taking a laxative or diuretic and/or excessive exercise, etc. to compensate for the binge. Bulimia nervosa also effects almost 90% of female. Unlike anorexia nervosa, people suffering from bulimia nervosa are usually normal or slightly over weight.

                    The Diagnosis



Diagnosis of bulimia nervosa is difficult, as people with Bulimia vervosa are very good in hiding the health problems and related symptoms, but certain signs of a person can be helpful.
A. Criteria
Based on the results of the clinical follow-up study of 41 female patients, diagnostic criteria of bulimia nervosa that should be used in clinical studies are suggested as follows:
(1) presence of anorexia nervosa or transitory amenorrhea in the premorbid period;
(2) eating attacks with losing of the control over food consumption not less than twice a week during 3 months;
(3) compensatory behavior in the form of spontaneous vomiting, abuse of purgative and diuretic medications etc;
(4) fear of obesity;
(5) cycloid affective changes with higher impulsivity, reduction of the control over primitive drives and/or expressed anxiety disorders; inclination to alcohol and drug abuse and nicotine dependence;
(6) changes of the body mass index;
(7) absence of amenorrhea. The disease dynamics is characterized by formation of the pathological cycle "diet--overeating--compensatory behavior" on the background of cyclothymic affective disorders. Two types of bulimia nervosa--with and without other drive disorders--have been singled out(25).

Others suggested that A diagnosis of bulimia nervosa is made when a person has recurrent episodes of binge eating, a feeling of lack of control over behavior during binges, regular use of self-induced vomiting, laxatives, diuretics, strict dieting, or vigorous exercise to prevent weight gain, a minimum of 2 binge episodes a week for at least 3 months, and persistent overconcern with body shape and weight. Patients with eating disorders are usually secretive and often come to the attention of physicians only at the insistence of others(25a).

B. Blood, urine tests and X ray
After taking the complex physical exam, including detail of absence of period and the examination the symptoms of Bulimia nervosa, Blood and urine tests may be ordered
a. Blood tests
The aim of the Blood tests are to check for signs of malnutrition, including levels of potassium levels and electrolyte imbalances.
b. Urine steroids
The increased level of the stress marker allo-tetrahydrocorticosterone refers to the involvement of stress in these diseases, but the relevance of hormone alteration to the pathophysiology of eating disorders remains to be elucidated(26)
c. X ray
The aim of the X ray to check for broken bones, pneumonia. In some cases, dual energy X-ray absorptiometry may be necessary to test for the presented osteopenia and osteoporosis(27)
d. Etc.

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Sources
(25) http://www.ncbi.nlm.nih.gov/pubmed/16841479
(25a)http://www.ncbi.nlm.nih.gov/pubmed/1475950
(26) http://www.ncbi.nlm.nih.gov/pubmed/15560936
(27) http://www.ncbi.nlm.nih.gov/pubmed/22137016


Wednesday, October 26, 2016

General Health: Eating Disorders - Bulimia nervosa - The Affects and Consequences

Kyle J. Norton(Scholar, Master of Nutrients), all right reserved.
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.


                               Eating disorders

Eating Disorders are defined as a group of abnormal eating habits associated to a person preoccupation weight, involving either insufficient or excessive food intake.

                                Bulimia nervosa

Bulimia nervosa is defined as a medical condition of consuming a large amount of food in a short amount of time or one setting (binge eating), followed by self induced vomiting, taking a laxative or diuretic and/or excessive exercise, etc. to compensate for the binge. Bulimia nervosa also effects almost 90% of female. Unlike anorexia nervosa, people suffering from bulimia nervosa are usually normal or slightly over weight.

                    The Affects and Consequences

A. Psychological effects
1. Mood disorders and suicidality
Onset of bulimia nervosa (BN) typically occurs in adolescence and is frequently accompanied by medical and psychiatric sequelae that may have detrimental effects on adolescent development. Potentially serious medical consequences and high comorbid rates of mood disorders and suicidality underscore the need for early recognition and effective treatments(14)

2. Substance abuse
In the study to examine the significance of a past history of substance abuse on treatment outcome for bulimia nervosa, showed that Although patients with a history of substance abuse reported higher levels of anxiety and depression at presentation for treatment than patients without such histories, the two groups reported a similar age of onset of their bulimia nervosa and similar severity of eating pathology with regards to binge and vomit frequencies and measures of concern about body shape and weight. On all outcome measures, the improvement of the substance abuse group was equal to or greater than that in the group without a history of substance abuse(15).

3. Etc.

A.2. Physical consequences
1. Acute gastric dilation
There are a report of a case of a young woman with bulimia nervosa who developed acute gastric dilation that was diagnosed by computerized tomography. The patient had no history of factors associated with delayed gastric emptying. The treatment course is reviewed, as is the pathophysiology of acute gastric dilation(16).


2. Electrolyte imbalances
Dr. Olson AF., in the study of Outpatient management of electrolyte imbalancesassociated with anorexia nervosa and bulimia nervosa, said "Bulimia nervosa and anorexia nervosa are eating disorders with significant morbidity that often go undetected. Nurses and primary care providers are encouraged to recognize the early signs and symptoms of these disorders and to intervene appropriately. Several case reports in this article describe patients with these disorders and various related electrolyte abnormalities. Understanding electrolyte imbalancesassociated with both disorders may lead to earlier effective intervention and overall improved health outcomes"(17).

2. Arrhythmias
In the examination of signal-averaged electrocardiography (SAECG). on 48 female ED patients [21 with anorexia nervosa (AN) and 27 with bulimia nervosa (BN)] and on 20 healthy women. An LP was judged positive if two or more of the following criteria were fulfilled: QRS duration >120 ms, root-mean-square voltage <20 microV, and a high-frequency, low-amplitude duration >38 ms. that indicated BN patients with a history of AN had significantly more SAECG abnormalities(18).


3. Oesophageal and gastric motor activity
In the study of esophageal and gastric motor activity in patients with bulimia nervosa, found that (i) bulimic behaviour can obscure symptoms of oesophageal motor disorders and (ii) gastric emptying is frequently delayed in bulimia nervosa(19).

4. Pancreatitis
There is a report of a 19-year-old woman with bulimia nervosa who died of acute hemorrhagic pancreatitis. The symptoms of both conditions are very similar, the pre-existence of an eating disorder should not distract physicians from the possibility that potentially lethal acute pancreatitis may coexist(20).

5. Absence of period
Some researchers suggested that amenorrhea is one of diagnostic criteria ofbulimia nervosa(21)

6. Visceral fat and increased adrenal gland volumes (AGV)
BN patients had significantly more visceral adipose tissue (VAT) (HC, 1589.3 +/- 967.6 ml versus 927.2 +/- 428.4 ml, p < .05) and an increased relative AGV (0.068% of body volume versus 0.048% of body volume, p < .05) compared with HC, although waist circumference and BMI did not differ. Although the VAT part in the upper abdomen was increased, especially the VAT of lower abdomen along with the pelvis or any subcutaneous fat compartment was not increased(22).

7. Sleep disturbance
Sleep disturbances are highly associated with anorexia nervosa (AN), buLimia nervosa (BN) and non-specified eating disorders (ED-NOS)(23)

8. Other physical effects
Dt, Mitchell JE and Crow S. at the University of North Dakota School of Medicine and the Neuropsychiatric Research Institute, in the study of Medical complications of anorexia nervosa and bulimia nervosa, showed that the frequently cited risk of premature death in those with anorexia nervosa. A plethora of dermatologic changes have been described, some signaling serious underlyingpathophysiology, such as purpura, which indicates a bleeding diathesis. Much of the literature continues to delineate the fact that diabetic patients with eating disorders are at high risk of developing diabetic complications.Gastrointestinal complications can be serious, including gastric dilatation andsevere liver dysfunction. Acrocyanosis is common, and patients with anorexianervosa are at risk of various arrhythmias. Low-weight patients are at high risk forosteopenia/osteoporosis. Nutritional abnormalities are also common, includingsodium depletion and hypovolemia, hypophosphatemia and hypomagnesemia. Resting energy expenditure, although very low in low-weight patients, increases dramatically early in refeeding(24).

Pregnancy Miracle
Reverse Infertility And Get Pregnant Naturally
Using Holistic Ancient Chinese Medicine

Sources
(14) http://www.ncbi.nlm.nih.gov/pubmed/22614677
(15) http://www.ncbi.nlm.nih.gov/pubmed/1502972
(16) http://www.ncbi.nlm.nih.gov/pubmed/17950174
(17) http://www.ncbi.nlm.nih.gov/pubmed/15785332
(18) http://www.ncbi.nlm.nih.gov/pubmed/16380317
(19) http://www.ncbi.nlm.nih.gov/pubmed/2323585
(20) http://www.ncbi.nlm.nih.gov/pubmed/15282695
(21) http://www.ncbi.nlm.nih.gov/pubmed/16841479
(22) http://www.ncbi.nlm.nih.gov/pubmed/19124623
(23) http://www.ncbi.nlm.nih.gov/pubmed/19630364
(24) http://www.ncbi.nlm.nih.gov/pubmed/16721178

Tuesday, October 25, 2016

General Health: Eating Disorders - Bulimia nervosa - The Causes and risk factors

Kyle J. Norton(Scholar, Master of Nutrients), all right reserved.
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.


                               Eating disorders

Eating Disorders are defined as a group of abnormal eating habits associated to a person preoccupation weight, involving either insufficient or excessive food intake.

                                Bulimia nervosa

Bulimia nervosa is defined as a medical condition of consuming a large amount of food in a short amount of time or one setting (binge eating), followed by self induced vomiting, taking a laxative or diuretic and/or excessive exercise, etc. to compensate for the binge. Bulimia nervosa also effects almost 90% of female. Unlike anorexia nervosa, people suffering from bulimia nervosa are usually normal or slightly over weight.

                    The Causes and risk factors

A. Causes
The causes of bulimia vervosa is unknown but in the study to examine the beliefs of women concerning causes and risk factors for eating-disordered behaviour, showed that having low self-esteem' was considered very likely to be a cause of BN by 75.0% of respondents, and the most likely cause by 40.5% of respondents. Other factors perceived as significant were 'problems from childhood', 'portrayal of women in the media', 'being overweight as a child or adolescent' and 'day-to-day problems', while genetic factors and pre-existing psychological problems were perceived to be of minor significance. Most respondents believed that women aged under 25 years were at greatest risk of having or developing BN(9).

B. Risk factors
1. Gender
It you are female, you are at 90% higher risk to develop bulimia nervosa.
2. Age
In both anorexia nervosa and bulimia nervosa, age at onset showed a significant decrease according to year of birth(10)
3. Social pressure
The fear of become fat due to wrongly influent in the western society where attractiveness is equal to thinness
4. Family history
If one the member of your direct family has bulimia nervosa, you are at increased risk to have that disease as well.
5. Migraine
Dr. D'Andrea G, and the research team at the Biochemistry Laboratory for the Study of Primary Headaches and Neurological Diseases, Research and Innovation S.p.A suggest that migraine may constitute a risk factor for the occurrence of ED in young females. This hypothesis is supported by the onset of migraine attacks that initiated, in the majority of the patients, before the occurrence of EDsymptoms, in the study of Is migraine a risk factor for the occurrence of eating disorders? Prevalence and biochemical evidences(11).

6. Obstetric complications
Researchers found that several complications, such as maternal anemia (P = .03), diabetes mellitus (P = .04), preeclampsia (P = .02), placental infarction (P = .001), neonatal cardiac problems (P = .007), and hyporeactivity (P = .03), were significant independent predictors of the development of anorexia nervosa. Therisk of developing anorexia nervosa increased with the total number of obstetric complications, the obstetric complications significantly associated withbulimia nervosa were the following: placental infarction (P = .10), neonatal hyporeactivity (P = .005), early eating difficulties (P = .02), and a low birth weight for gestational age (P = .009). Being shorter for gestational age significantly differentiated subjects with bulimia nervosa from both those with anorexia nervosa (P = .04) and control subjects (P = .05)(12).

7. DRD4 gene
Although there is no evidence of the direct association between DRD4 gene andbulimia nervosa, researchers suggested that its variants are associated with a history of childhood ADHD in BN probands. This may have relevance for the understanding, prevention, and treatment of BN that evolves in the context of childhood ADHD(12a).

8. Other risk factors includes
Low self-esteem', 'problems from childhood', 'portrayal of women in the media', 'being overweight as a child or adolescent' and 'day-to-day problems', while genetic factors and pre-existing psychological problems(13)

9. Etc.

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Sources
(9) http://www.ncbi.nlm.nih.gov/pubmed/15209840
(10) http://www.ncbi.nlm.nih.gov/pubmed/20141711
(11) http://www.ncbi.nlm.nih.gov/pubmed/22644175
(12) http://www.ncbi.nlm.nih.gov/pubmed/16389201
(12a) http://www.ncbi.nlm.nih.gov/pubmed/22271608
(13) http://www.ncbi.nlm.nih.gov/pubmed/15209840